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English: Cancer-associated thrombosis can result from: (1) stasis, i.e., direct pressure on blood vessels by the tumor mass, poor performance status, and bed rest following surgical procedures; (2) iatrogenic, due to treatment with antineoplastic medications; and (3) secretion of heparanase from malignant tumors that results in degradation of endogenous heparin.
Date
Source Potential Mechanisms of Cancer-Related Hypercoagulability. Nasser NJ, Fox J, Agbarya A. Cancers (Basel). 2020 Feb 29;12(3):566. https://doi.org/10.3390/cancers12030566
Author Nicola J. Nasser, Jana Fox, and Abed Agbarya

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current23:26, 19 February 2023Thumbnail for version as of 23:26, 19 February 20231,999 × 1,135 (794 KB)Jul059Lossless file size reduction
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