List of causes of shortness of breath
(Redirected from Differential diagnosis of shortness of breath)
Many different conditions can lead to the feeling of dyspnea (shortness of breath). DiagnosisPro, an online medical expert system, listed 497 in October 2010.[1] The most common cardiovascular causes are acute myocardial infarction and congestive heart failure while common pulmonary causes include: chronic obstructive pulmonary disease, asthma, pneumothorax, and pneumonia.[2]
Pulmonary
[edit]- Obstructive lung diseases
- Diseases of lung parenchyma and pleura
- Contagious
- Anthrax through inhalation of Bacillus anthracis
- Pneumonia
- COVID-19
- Non-contagious
- Contagious
- Pulmonary vascular diseases
- Acute or recurrent pulmonary emboli
- Pulmonary hypertension, primary or secondary
- Pulmonary veno-occlusive disease
- Superior vena cava syndrome
Other causes
[edit]- Obstruction of the airway
- Immobilization of the diaphragm
- Lesion of the phrenic nerve
- Polycystic liver disease
- Tumor in the diaphragm
- Restriction of the chest volume
- Ankylosing spondylitis
- Broken ribs
- Kyphosis of the spine
- Obesity
- Costochondritis
- Pectus excavatum
- Scoliosis
- Disorders of the cardiovascular system
- Disorders of the blood and metabolism
- Disorders of the neurological system
- Ischemic stroke (when it occurs in the brain stem)
- Brain bleed (when it occurs in the brain stem)
- Disorders affecting breathing nerves and muscles
- Psychological conditions
- Medications
- Other
References
[edit]- ^ "Differential Diagnosis For Dyspnea: Poisoning (Specific Agent)". Archived from the original on 2010-11-16. Retrieved 2012-08-23.
- ^ Shiber JR, Santana J (May 2006). "Dyspnea". Med. Clin. North Am. 90 (3): 453–79. doi:10.1016/j.mcna.2005.11.006. PMID 16473100.
- ^ Hwu WL, Suzuki Y, Yang X, et al. (February 2000). "Late-onset holocarboxylase synthetase deficiency with homologous R508W mutation". J. Formos. Med. Assoc. 99 (2): 174–7. PMID 10770035.
- ^ Simpson, Kathleen Rice; Patricia A Creehan (2007). Perinatal Nursing (3rd ed.). Lippincott Williams & Wilkins. p. 66. ISBN 978-0-7817-6759-0.